You are here

  • Home
  • Archive
  • Volume 69, Issue 3
  • Hormesis and public health: can glutathione depletion and mitochondrial dysfunction due to very low-dose chronic exposure to persistent organic pollutants be mitigated?

Article info

Article menu
Download PDFPDF
Review
Hormesis and public health: can glutathione depletion and mitochondrial dysfunction due to very low-dose chronic exposure to persistent organic pollutants be mitigated?

Authors

  • Duk-Hee Lee Department of Preventive Medicine, School of Medicine, Kyungpook National University, Daegu, Korea Department of Biomedical Science, BK21 Plus KNU Biomedical Convergence Program, Kyungpook National University, Daegu, Korea PubMed articlesGoogle scholar articles
  • David R Jacobs Division of Epidemiology, School of Public Health, University of Minnesota, Minneapolis, Minnesota, USA PubMed articlesGoogle scholar articles
  1. Correspondence to Duk-Hee Lee, Department of Preventive Medicine, School of Medicine, Kyungpook University, 101 Dongin-dong, Jung-gu, Daegu 700-422, Korea; lee_dh{at}knu.ac.kr
View Full Text

Citation

Lee D, Jacobs DR
Hormesis and public health: can glutathione depletion and mitochondrial dysfunction due to very low-dose chronic exposure to persistent organic pollutants be mitigated?
J Epidemiol Community Health 2015;69:294-300.

Publication history

  • Received June 16, 2014
  • Revised September 1, 2014
  • Accepted September 7, 2014
  • First published September 30, 2014.
Online issue publication 
April 27, 2016

Article Versions

Request permissions

If you wish to reuse any or all of this article please use the link below which will take you to the Copyright Clearance Center’s RightsLink service. You will be able to get a quick price and instant permission to reuse the content in many different ways.

Copyright information

Published by the BMJ Publishing Group Limited. For permission to use (where not already granted under a licence) please go to http://group.bmj.com/group/rights-licensing/permissions