EDITORIAL
Childhood learning and behavioural disorders
Invited commentary: persistent organic pollutants and childhood learning and behavioural disorders
1 Department of Medicine, Channing Laboratory, Brigham and Women’s Hospital, Harvard Medical School, Boston, Massachusetts, USA
2 Department of Environmental Health, Harvard School of Public Health, Boston, Massachusetts, USA
Correspondence to:
Correspondence to:
Dr S A Korrick
Channing Laboratory, 181 Longwood Avenue, Boston, MA 02115, USA; susan.korrick@channing.harvard.edu
Association of learning disabilities and attention-deficit disorder with concurrent levels of persistent organic pollutants
| The first 150 words of the full text of this article appear below. |
In this issue of Journal of Epidemiology & Community Health, Lee et al1 (see page 591) demonstrate an association of both learning disabilities (LD) and attention-deficit disorder (ADD) with concurrent serum levels of persistent organic pollutants (POPs) among 12–15-year-old participants in the National Health and Nutrition Examination Survey (NHANES 1999–2000). For example, among children with detectable versus non-detectable levels of heptachlorodibenzo-p-dioxin (HPCDD), the adjusted prevalence odds ratios (95% CI) for LD and ADD were 2.08 (1.17 to 3.68) and 3.41 (1.08 to 10.8), respectively. This is perhaps the first published study to describe a potential POP-associated increased risk of LD or ADD. Previous studies have primarily assessed continuous cognitive or behavioural measures among generally healthy populations. LD and ADD are clinical end points that have obvious public health impacts in terms of childhood (and adult) morbidity as well as educational,
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